Meet Stephen F. Calderon, M.D.

Stephen F. Calderon, M.D. is a senior attending physician and chairman of the department of Neurological Surgery at Saint Francis Hospital & Medical Center in Hartford, CT. His specialties include Spinal Surgery, Neurovascular Surgery, and Neuro-oncology. He is licensed to practice in the state of Connecticut and has received numerous “Top Doctor” awards in Connecticut and Hartford Magazines.

Dr. Calderon is part of the Spine Institute of Connecticut at Saint Francis Hospital.  This has promoted collaborative review of challenging cases as well as participation in a national databank to track and analyze outcomes.  This has resulted in very effective management of patients with spine problems.  All surgical procedures are performed in a state-of-the-art operating room with the latest technology.

Dr. Stephen Calderon maintains surgical privileges at several area hospitals.  This section lists affiliated hospitals with links to their websites:

• St. Francis Hospital
• Hartford Hospital
• Connecticut Surgery Center
• Middlesex Hospital

Undergraduate Degree
Washington and Jefferson College
Washington, PA
B.A. 1978
Phi Beta Kappa Graduate

Medical Degree
University of Rochester School of Medicine and Dentistry
Rochester, New York
M.D. 1982

Internship, General Surgery University Hospital, University of Cincinnati Cincinnati, OH 1982-1983Internship, Neurological Surgery The Christ Hospital Cincinnati, OH 1983-1984Residency, Neurological Surgery Children’s Hospital Medical Center Cincinnati, OH 1984-1985Residency, Neurological Surgery Veterans Administration Medical Center University of Cincinnati College of Medicine Cincinnati, OH 1985-1986

Residency, Neurological Surgery Good Samaritan Hospital Cincinnati, OH 1985-1987Chief Resident of Neurosurgery University Hospital University of Cincinnati Cincinnati, OH 1988Chief Resident of Neurosurgery Children’s Hospital Medical Center Cincinnati, OH 1988Chairman, Department of Neurological Surgery St. Francis Hospital, Hartford, CT September 1996 – Present

St. Francis Hospital and Medical Center Attending Physician, Department of Neurosurgery Hartford, CT 06105 1988 – PresentManchester Memorial Hospital Attending Physician, Department of Neurosurgery Manchester, CT 06040 ?- PresentRockville General Hospital Attending Physician, Department of Neurosurgery Rockville, CT 06066 ? – PresentNew Britain General Hospital Affiliated Neurosurgeon New Britain, CT 06051 ? – Present

Middlesex Hospital Attending Physician, Department of Neurosurgery Middlesex, CT 06457 ? – 2003Bristol Hospital Attending Physician, Department of Neurosurgery Bristol, CT 06010 ? – 2003Hartford Hospital Courtesy Neurosurgeon Hartford, CT 06106 1999 – Present

• American Association of Neurological Surgeons 
• Congress of Neurological Surgeons 
• American Medical Association 
• Hartford County Medical Association 
• Connecticut State Medical Association

Research Papers Presented at Scientific Meetings:

“Increased dopaminergic sensitivity following prenatal MPTP and MPT+ treatments in rats.” Society for Neuroscience, New Orleans, Louisiana. Sanberg PR, Garver DL, Norman AB, Calderon SF. November 10-14, 1987 

“A new rotational behavior model for assessing the restructuring of striatal dopamine effector systems: Are transplants sensitive to peripherally acting drugs?” Schmitt Symposium, Rochester, NY. Norman AB, Giordano M, Sanberg PR, Calderon SF. June 29-July 3, 1987 

“The quinolinic acid model of Huntington’s Disease: Dose-dependent effects on behavior.” Society for Neurosciences, New Orleans, Louisiana. Giordano M, Norman AB, Sanberg PR, Calderon SF. November 10-14, 1987 

“Striatal transplants promote centrally-mediated behavioral effects of N-methyl-scopolamine which does not normally cross the blood-brain barrier.” Society for Neurosciences, New Orleans, Louisiana. Sanberg PR, Weissman EM, Giordano M, Norman AB, Calderon SF. November 10-14, 1987 

“Attention of apomorphine-induced rotational behavior by fetal striatal tissue transplants in rats with unilateral striatal kainic acid lesions.” Society for Neurosciences, New Orleans, Louisiana. Norman AB, McGowan T, Giordano M, Sanberg PR, Calderon SF. November 10-14, 1987 

Research Publications:

Scott DE, Gash DM, Sladek JR, Clayton C, Mitchell JA, Calderon SF, Paull WK: Organization of the Mammalian Cerebral Ventricular System: Ultrastructural Correlates of CSF – Neuropeptide Secretion. Frontiers in Hormone Research, 9:15-35, 1982 

Sanberg PR, Calderon SF, Garver DL, Norman AB: Brain Tissue transplants in an animal model of Huntington’s Disease. Psychopharmacology Bulletin, 23:476-482, 1987 

Norman AB, Calderon SF, Giordana M, Sanberg PR: Striatal tissue transplants attenuate apomorphine-induced rotation behavior in rats with unilateral kainic acid lesions. Neuropharmocology, 27:333-336, 1988 

Norman AB, Calderon SF, Giordano M, Sanberg PR: A novel rotational behavior model for assessing the restructuring of striatal dopamine effector systems: Are transplants sensitive to peripherally acting drugs? Progress in Brain Research, in press 

Calderon SF, Sanberg PR, Norman AB: Quinolinic acid lesions of rat striatum abolish DL dopamine receptor mediated catalepsy. Brain Research, in press 

Abstracts:

Sanberg PR, Norman AB, Calderon SF, Garver DL: Neural implants in an animal model of Huntington’s disease. Society of Biological Psychiatry, 42:161, 1987 

Norman AB, Calderon, SF, Giordano M, Sanberg PR: A novel rotational behavior model for assessing the restructuring of striatal dopamine effector systems: Are transplants sensitive to peripherally acting drugs? Schmitt Neurological Sciences Symposium, 1987 

Norman AB, McGowan T, Calderon SF, Giordano M, Sandberg PR: Attenuation of apomorphine-induced rotational behavior by fetal striatal tissue transplants in rats with unilateral striatal kainic acid lesions. Society for Neuroscience Abstracts, 13:1602, 1987 

Calderon SF, Sanberg PR, Garver DL, Norman AB: Increased dopaminergic sensitivity following prenatal MPTP and MPP+ treatments in rats. Society for Neuroscience Abstracts 13:1602, 1987 

Giordano M, Calderon SF, Giordano M, Weissman EM, Norman AB: The quinolinic acid model of Huntington’s disease: Dose-dependent effects on behavior. Society for Neuroscience Abstracts, 13:1360, 1987 

Sanberg PR, Calderon SF, Giordano M, Weissman EM, Norman AB: Striatal transplants promote centrally mediated behavioral effects of N-methylscopolamine which does not normally cross the blood-brain barrier. Society for Neuroscience Abstracts, 13:1360, 1987 

Sanberg PR, Calderon SF, Giordano M, Weissman EM, Zubrycki EM, Garver DL, Norman AB: Pharmacological recovery of dopaminergic neurotransmission following fetal striatal transplants in an animal model of Huntington’s disease. Proceedings of the American College of Neuropsychopharmacology, 1987 

Norman AB, Giordano M, Calderon SF, Weissman EM, Chopra YM, Sanberg PR: Striatal tissue transplants reverse functional deficits in an animal model of Huntington’s disease. Neurology 38 (Supplement):203, 1988 

Calderon SF, Norman AB, Giordano M, Weissman EM, Shipley MT, Sanberg PR: The blood-brain barrier surrounding striatal transplants is permeable to peripherally acting drugs. Neurology 38 (Supplement):145, 1998 

Weissman EM, Sanberg PR, Calderon SF, Zubrycki EM, Norman AB: Long-term effects of prenatal MPTP treatment on the development of rat dopaminergic systems. Neurology 38 (Supplement):230, 1988 

Giordano M, Calderon SF, Norman AB, Sanberg PR: Quinolinic acid lesions of the striatum produce behavioral model of Huntington’s disease. Neurology 38 (Supplement):329, 1988 

Weissman EM, Norman AB, Calderon SF, Zubrycki EM, Sanberg PR: Dopamine system development: The effect of prenatal MPTP on dopamine medicated behavior. American Psychiatric Association

Current Research Activities:

Delineation of behavioral, neurochemical, pharmacologic properties of homologous fetal brain transplants in a rat model of Huntington’s disease.